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The schedule is as follows: day 1: edema, diameter and number of blood vessels, number of inflammatory cells and reticulin, and number of vital follicles

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These observations suggest that aminochrome is involved in the alpha synuclein-dependent inhibition of CMA autophagy because aminochrome induces the formation of -synuclein protofibrils, such as the A53T mutant (Figure 10)

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